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Oesophageal pathology

Key points


Congenital oeosphageal stenosis


  1. Mucosal webbing 16% - Upper/Middle 1/3

  2. Fibromuscular hypertrophy 54% - Middle/Lower 1/3

  3. Tracheobronchial rests and remnants 30% - Lower 1/3


Failure of tracheo-oesophageal separation

Possibly on the same spectrum as OA/TOF


Web + iron deficiency - Plummer-Vinson/Paterson-Brown-Kelly

Iron deficiency leads to loss of iron dependent enzymes + mucosal degeneration

Associated with autosomal recessive form of epidermolysis bullosa


Investigations

UGI contrast

Manometry to differentiate from Achalasia

Iron studies for web

Endoscopy - web will be thin eccentric lesion, normal mucosa


Management

Trial of dilatations

95% success rate for web

Unlikely to be successful in TBR


Segmental oesophagectomy if dilatations fail

Thoracoscopy or thoracotomy

Identify level with on table endoscopy

Preserve vagus nerves

TAT tube

Persistent symptoms in 50% after procedure


Oesophageal diverticula


  1. Pharyngoesophageal - Zenker - between cricopharyngeus + inferior constrictor - pulsion - false diverticulum lacking outside muscle

  2. Midoesophageal - complication of histoplasmosis/TB/sarcoid/lymphoma, inflammation causes external traction - true diverticula containing all oesophageal wall layers

  3. Epiphrenic - pulsion - false diverticulum lacking outside muscle - right posterior wall - secondary to functional obstruction e.g., Achalasia, fundoplication


Also can be acquired as a result of OA/TOF


Investigations

Contrast

Endoscopy


Management

Pharyngoesophageal diverticulum - Cricopharyngeal myotomy + resection

Midoesophageal - Right thoracotomy, adhesiolysis, resection + closure in layers + intercostal flap

Epiphrenic - Left thoracotomy + stapled resection + myotomy on opposite side


Oesophageal replacement


2 most common indications:

Long gap OA - should do at 3 months if failed anastomosis (ERNICA)

Corrosive stricture


Ideal replacement:


Allows normal feeding

Permits replacement of the entire oesophagus when indicated

Does not cause respiratory compromise

Does not become tortuous or redundant

Gastro-oesophageal reflux in the conduit should be minimal

Does not have increased malignant risk

Should function normally for the life-time of the individual (i.e. over 70 years)


Gastric - most common

Should bring up through mediastinum - 2 incisions - abdomen and cervical - do not open thorax, do not perform thoracic anastomosis

Thoracic stomach will be dysmotile

Can treat with domperidone

Do pyloroplasty (Recommended by ERNICA)

Do jejunostomy

If dilated - thoracic gastroplasty


Other options:

Gastric tube (use greater curvature and rotate up)

Jejunum - cannot do if has oesophagostomy

Right hemicolon - isoperistaltic (ERNICA) - need to do fundo and pyloroplasty as well



Page edited by Mrs Charnjit Seehra BSc November 2024


References


Jensen, Amanda R, et al. "Congenital Esophageal Stenosis." Pediatric Surgery NaT, American Pediatric Surgical Association, 2020. Pediatric Surgery Library, www.pedsurglibrary.com/apsa/view/Pediatric-Surgery-NaT/829675/all/Congenital_Esophageal_Stenosis.


Trappey, Francois , et al. "Esophageal Diverticula." Pediatric Surgery NaT, American Pediatric Surgical Association, 2020. Pediatric Surgery Library, www.pedsurglibrary.com/apsa/view/Pediatric-Surgery-NaT/829745/all/Esophageal_Diverticula.


Dingemann C, et al. ERNICA Consensus Conference on the Management of Patients with Long-Gap Esophageal Atresia: Perioperative, Surgical, and Long-Term Management. Eur J Pediatr Surg. 2021 Jun;31(3):214-225. doi: 10.1055/s-0040-1713932. Epub 2020 Jul 15. PMID: 32668485.


Arul GS, Parikh D. Oesophageal replacement in children. Ann R Coll Surg Engl. 2008 Jan;90(1):7-12. doi: 10.1308/003588408X242222. PMID: 18201490; PMCID: PMC2216706.

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Please note that all information on this site is for professional educational purposes only, it does not constitute medical advice

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